Clinical Manifestations


Sudden death
  • may be the presenting clinical manifestation of coronary atherosclerosis, but is more likely to occur in patients with prior myocardial infarction and moderate-to-severe left ventricular dysfunction
  • approximately 20% of patients with acute myocardial infarction die before reaching a hospital (unattended death)
  • ventricular fibrillation is responsible for most cases of sudden death
Angina pectoris
  • angina pectoris is a clinical condition characterised by precordial chest pain, usually precipitated by exertion or stressful conditions and rapidly relieved by rest or nitrate administration
  • the diagnosis is established on the basis of electrocardiographic or scintigraphic evidence of ischaemia during pain. When angiography is performed for the sake of diagnosis, evidence is usually provided of significant obstruction of major coronary vessels
  • angina is generally caused by the presence of atherosclerotic lesions, producing flow-reducing stenoses
  • coronary vasospasm may be associated with arterial lesions or, less frequently, may occur in apparently normal vessels
  • angina may be part of the clinical picture of severe myocardial hypertrophy, severe aortic stenosis or insufficiency, hyperthyroidism, pronounced anaemia or paroxysmal tachycardias, even in the absence of haemodynamically significant coronary stenosis
Unstable angina
  • the term 'unstable angina' refers to a condition of clinical deterioration of a pain pattern in patients with previously stable angina
  • new-onset angina is also considered unstable
  • coronary evaluation of patients with unstable angina demonstrates in most cases the occurrence of complex coronary stenoses, suggesting plaque rupture and thrombosis [Fuster, 1999]
  • this unstable condition may progress to complete occlusion and acute myocardial infarction, or might improve and evolve towards a pattern of stable, although possibly more severe, angina
Acute myocardial infarction
  • acute myocardial infarction is characterised by sudden and prolonged anterior or occasionally posterior chest discomfort, often in association with arrhythmia, hypotension, shock or cardiac failure
  • typical electrocardiographic abnormalities and/or elevation of cardiac enzymes confirm the clinical diagnosis. At echocardiography, segmental wall motion abnormalities are usually detectable
  • in most instances, this severe condition is produced by an occlusive coronary thrombus at the site of a pre-existing atherosclerotic lesion [Fuster, 1999]
  • the severity of the clinical picture and the prognosis are, to a large extent, related to the size and anatomical location of the myocardial infarction
  • extension of myocardial damage may occur in the case of impaired collateral circulation or further thrombotic events in the adjacent coronary arteries
  • life-threatening complications of myocardial infarction include post-infarction ischaemia, arrhythmia, myocardial dysfunction and ventricular failure, cardiogenic shock, rupture of papillary muscles or of the inter-ventricular septum, myocardial rupture and left ventricular aneurysm
Cerebral infarction
  • cerebral infarction caused by atherosclerosis and thrombosis of cerebral vessels represents a significant fraction of the cases of stroke
  • a frequent cause of obstruction of the cerebral vessel is represented by emboli derived from ulcerated plaques on a major artery to the brain
  • atherosclerotic plaques often occur in the region of carotid bifurcation extracranially [Iannuzzi, 1995]
  • cerebral infarction might also be produced by emboli of cardiac origin and is associated with rheumatic heart disease, mitral valve disease, arrhythmia and mural thrombi complicating an acute myocardial infarction. The resulting neurological deficit depends upon the particular vessel involved and the extent of compensation via the collateral cerebral circulation
Transitory ischaemic attack
  • the condition of a focal ischaemia causing a neurological deficit that lasts for less than 24 hours is called a transitory ischaemic attack
  • a transitory ischaemic attack is generally caused by emboli in the cerebral circulation and often occurs without leading to cerebral infarction (as a result of spontaneous fibrinolysis or effective collateral circulation)
  • approximately 30% of patients with stroke have a history of transitory ischaemic attacks
Aortic and peripheral atherosclerosis
  • atherosclerotic disease of the aorta and its branches (peripheral atherosclerosis) is a major cause of disability
  • obliterating arterial disease of the lower limbs (in some cases manifested as intermittent claudication), mesenteric ischaemia leading to recurrent abdominal pain or renal ischaemia associated with hypertension are the consequences of thrombosis complicating atherosclerotic plaques
  • abdominal aortic aneurysms (arterial dilatation complicating severe atherosclerosis) may be asymptomatic for years and are most often diagnosed when an echography of the abdomen is undertaken for other reasons
  • abdominal aortic aneurysms manifest themselves at the time of the rupture with back or abdominal pain and carry a very poor prognosis if elective surgical resection is not performed before rupture


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